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Glutamate undergoes deamination, an oxidative reaction catalysed by glutamate dehydrogenase, [18] as follows: Ammonia (as ammonium) is then excreted predominantly as urea, synthesised in the liver

Exendin-4 administration reduced visceral hypersensitivity in a dose-dependent manner and decreased serotonin levels in the colon

Pharmacological modulation of cortisol levels, combined with interventions to regulate glutamate clearance and recycling, may help restore neuronal homeostasis and reduce excitotoxicity

79 The promising benefits observed in major cardiovascular trials have sparked interest in their potential application for HF, particularly in patients with comorbid T2D or metabolic syndrome